Lange, Karen Suleman, Aly Srayko, Martin Supplemental Material for Lange, Suleman, and Srayko, M., 2019 p.p1 {margin: 0.0px 0.0px 0.0px 0.0px; font: 11.0px Arial} <p>Figure S1 shows anti-Ska immunofluorescence in one-cell embryos treated with control or ska-1 RNAi. Figure S2 shows KLP-7-GFP in the one-cell embryo is not altered by ska-1(RNAi) or ska-3(RNAi). Figure S3 shows Ska mutations do not restore microtubule levels in rsa-1(or598) embryos. Figure S4 shows TPXL-1-GFP at the centrosome is not altered by ska-1(RNAi). Figure S5 shows Ska is required to form a tight metaphase plate. Figure S6 shows the rsa-1(or598) spindle-collapse phenotype is partially rescued by loss of Ska function. Figure S7 shows the rsa-1(or598) spindle-collapse phenotype is partially rescued by loss of KMN kinetochore components and loss of EBP-2. Table S1 contains information on C. elegans strains used in this study. Table S2 contains information on cloning primers used in this study.</p> PP2A phosphatase;Ska complex;C. elegans;centrosome;kinetochore;microtubule;Aurora kinase;TPX2;Spindle assembly;Cell Biology 2019-04-24
    https://gsajournals.figshare.com/articles/dataset/Supplemental_Material_for_Lange_Suleman_and_Srayko_M_2019/8031848
10.25386/genetics.8031848.v1